An Anecdotal Overview on Psychological Effects of Receptor Activity
Roughly knowing how some receptors influence the mind and by correlating effects of medication and other substances over time, I experienced many combinations of these states and became increasingly aware of which receptor activity causes specific behavioral and psychological traits. I dont know enough to say whether the picture im drawing is accurate.
Dopamine
Anecdotal/Empirical Experiences
- Dopamine signaling influences thought structure, reward, impulsive, and addictive behavior.
- Increased D1 activity seems to increase impulsivity, though it’s hard to directly link to specific behaviors.
- Increased limbic D2 activity feels like it speeds up thoughts, causing racing thoughts, paranoia, anxiety, and sometimes delusional thinking.
- Prolonged high D2 activity in the mesolimbic pathway can lead to psychosis or schizophrenia-like symptoms.
- Increased D3 activity intensifies craving, greed, compulsive, and drug-seeking behavior.
- Blocking D2 activity with medications like Amisulpride can reduce psychotic symptoms, depending on dose and severity.
Theoretical/Broad Scientific Consensus
- Dopamine is strongly linked to motivation, reward, and reinforcement learning.
- Excess dopamine in certain brain areas is associated with psychosis, while low dopamine is linked to lack of motivation and motor symptoms (e.g., Parkinson’s disease).
- D3 receptors are especially prominent in the limbic system and are associated with motivation, craving, and compulsive or drug-seeking behavior.
- Dopamine antagonists (antipsychotics) are standard treatments for schizophrenia and related disorders.
Serotonin
Anecdotal/Empirical Experiences
- Serotonin influences mood; higher levels make you feel calm and comfortable.
- 5HT2A activation (e.g., by LSD or psilocin) alters perception and thought structure, and can sometimes cause panic or erratic thinking.
- 5HT2C activation also contributes to psychedelic effects and may affect thought patterns.
- 5HT3 seems to reduce erratic pain signaling.
- 5HT1A might influence NMDA (glutamate) signaling.
- 5HT4 (in the thorax) appears to boost cAMP levels.
- Strong serotonin hyperactivity can induce psychosis, possibly through 5HT1A/2C.
- Broad serotonin increases (like with SSRIs) usually elevate mood and create feelings of comfort and tranquility.
Theoretical/Broad Scientific Consensus
- Serotonin is a key regulator of mood, anxiety, and perception.
- 5HT2A activation is responsible for the psychedelic effects of drugs like LSD and psilocybin.
- SSRIs (which increase serotonin) are widely used to treat depression and anxiety.
- Excessive serotonin activity can contribute to serotonin syndrome, with symptoms including agitation, confusion, and hallucinations.
Norepinephrine (ADHD)
Anecdotal/Empirical Experiences
- Norepinephrine helps people with ADD/ADHD feel more focused and productive, reducing chaotic and disorganized thought patterns.
- High norepinephrine makes you alert and triggers a fight-or-flight response, especially if serotonin is low.
Theoretical/Broad Scientific Consensus
- Norepinephrine is involved in attention, arousal, and stress response.
- Medications that increase norepinephrine (like stimulants) are standard treatments for ADHD.
- High norepinephrine levels are associated with increased alertness and anxiety.
Hypocretin (Orexin)
Anecdotal/Empirical Experiences
- High hypocretin activity makes it impossible to fall asleep, even if you’re tired or other sleep-promoting systems are active.
Theoretical/Broad Scientific Consensus
- Hypocretin (orexin) is essential for wakefulness and arousal.
- Deficiency in hypocretin leads to narcolepsy; excess activity is linked to insomnia and difficulty initiating sleep.
Histamine
Anecdotal/Empirical Experiences
- Blocking histamine makes you feel tired and drowsy.
Theoretical/Broad Scientific Consensus
- Histamine promotes wakefulness; antihistamines (H1 blockers) are sedating and commonly used as sleep aids.